In its edition of July 15, 2011 Le Monde published an article under the title of "Murdoch: au Royaume-Uni, la peur a changé de camp", an article in which the events surrounding the demise of the Murdoch empire are described as "the UK, living its after-Murdoch spring". It is the first article that I have seen which draws an explicit parallel between the Arab Spring and events surrounding the Murdoch empire, although there may of course have been others, and concludes that there was a common factor - loss of fear.
The point being made here is that British politicians, fearful of tackling what they have perceived as the power of the Murdoch press, have suddenly lost their fear. The article does not mention another important similarity with the Arab Spring: the emotional trigger. Just as the Arab Spring had one(s), so the emotional trigger in this instance, I think, was the revelation that journalists working for one of the Murdoch papers had hacked into the mobile 'phone of a murdered girl and had deleted messages on her 'phone, thus giving false hopes to her family that she may have been alive when the search for her was on. The public was revolted. The trigger was ignited; there was no turning back, and the amygdala in the brains of politicians was de-activated, with consequences that we now know - the demise of the News of the World, plus other events that are waiting to happen.
Then came the revelation that the mobile 'phones of relatives of soldiers killed in Afghanistan may also have been hacked. I daresay that should the current reported investigations in the USA prove that the mobile 'phones of the victims of 9/11 had been hacked, the emotional volcano will be too hot to contain. This is an extremely emotive issue, and the consequences will be dire.
Although there have been some remarkable Members of Parliament who have fearlessly attacked the Murdoch press, the report in Le Monde and other reports in the British press have consistenly written that politicians here were dead scared of saying anything that may upset the Murdoch press and cost them their jobs and imperil their future rise. But once the emotional trigger was set alight, that fear was lost, presumably through amygdalar de-activation.
Another similarity with the Arab Spring, is that this was a mass event, in the sense that the revulsion at the revelations was widespread. Hence, an aid in the loss of fear was the emotional support of a wide segment of the public (see my previous post).
Several articles have emphasized the fact that maybe Murdoch did not have as much as power as he was assumed to have, and that he was only perceived to have this power. The point is really largely academic; power is always the power that is perceived, by the brain of course, never the real one which is in any case difficult to calculate.
And the brain may not be far off the mark. After all, when the British Prime Minister is reported as having had no less than 25 meetings with the Murdochs since coming to power, enjoyed a Christmas dinner with them, and when a previous Prime Minister, Tony Blair, is reported to have had three telephone conversations with Rupert Murdoch before the launch of the Iraq war, what else can the brain conclude?
Add to that the fact the the Business Secretary, Vince Cable, was removed from his position as chairman of a committee to decide the total Murdoch take-over of BSkyB, when he declared - long before these events reached boiling point - that he was "at war" with the Murdoch empire, what else can the brain conclude?
Such events, as the Daily Telegraph said in its editorial yesterday, "would have raised eyebrows in Palermo".
The only reality we have is brain reality - and brain reality seems to have been pretty shrewd at making inferences about power.
So, the amygdala is finally de-activated among a large number of members of that sheepish organization known as the Mother of Parliaments. Papers here are speaking about Parliament finding a role and purpose again. I rather doubt it. Which brings me back to the point: how long does amygdalar de-activation last? An interesting experimental question which may lead to lasting new views about politicians.
Showing posts with label the brain. Show all posts
Showing posts with label the brain. Show all posts
Sunday, July 17, 2011
The Arab Spring, Twitter, Facebook and the brain
A great deal has been written about what has come to be known as the “Arab Spring”. Two factors have reportedly been instrumental in enabling it. One of these is hardly credible, at least to me; the other is of profound neurobiological interest.
The first is that this is a social network media revolution, through channels such as Facebook and Twitter. I find it hard to credit this oft-repeated belief. Both Tunisia (where the revolt started) and Egypt are very poor countries, 40% of the population in the latter living below the beltline of $2 per day. I do not suppose that, apart from a tiny percentage, many have the means to acquire computers and mobile ‘phones. Indeed it is hard to believe that Mohammad Bouazizi, the young man who tragically and in despair over his poverty immolated himself, had the means to have access to social media networks at all. Television, commonly available in cafés for communal watching, is likely to have played a more significant role. This is not to say that Twitter and Facebook did not facilitate communication. Of course they did, just as (in a much slower world) the horse facilitated communication during the French Revolution.
The second – loss of fear - is of greater significance and of much neurobiological interest. We have been repeatedly told that the masses who are revolting, commonly against much better equipped security forces, have lost their fear. Fear is associated with certain physiological activity, and especially prominent among brain structures contributing to such activity is a complex nucleus called the amygdala, buried within the temporal lobes of the brain, and consisting of many subdivisions. I would not wish to imply that the amygdala alone is responsible for so complex a state, for the amygdala is connected to many other brain structures which, collectively, are responsible for generating and maintaining the state of fear, as a defensive mechanism to protect the individual. Whatever the role of the different brain structures, the central role played by the amygdala was shown many years ago when scientists described how damage to it results in a loss of fear by animals and humans.
The amygdala has extensive connections within the brain. It is believed that there are two routes to the amygdala, an “immediate” one from the sense organs, which by-passes the cerebral cortex, and a more “leisurely” one that relays signals through the cerebral cortex. The amygdala is also connected to centres, such as those of the sympathetic nervous system, which regulate activity to mobilize the individual for appropriate reaction in response to fearful events or stimuli.
I suppose that the default state is activity – whatever its exact nature - within this system, activity within the amygdala that is relayed to other centres with which it is connected. I also assume, perhaps somewhat simplistically, that de-activation of this system is what leads to the condition that we describe as “loss of fear”.
This raises the interesting question of what triggers the de-activation and what dictates how long-lasting the de-activation and therefore the change from the default state is.
It is obvious that a long set of grievances reaches a point where individuals defy willfully the physiological state and care no longer about the consequences of their action. In the case of the Arab Spring, there was also an emotional trigger – the self immolation by Mohammad Bouazizi. The amygdala is part of the brain’s emotional and social system and the relative influence of the emotional component compared to the more cognitive component in regulating its activity is interesting to determine.
I am of the view that, in Egypt, a trigger, which was far more significant than Twitter and Facebook messages, was the emotional breakdown of Wael Ghoneim on Egyptian TV, an episode that was played and replayed, and presumably seen by masses in the many cafés in Egyptian cities. The episode was actually accompanied by music, hence fortifying the emotional message.
The fear system has been studied largely in animals, but the events of the Arab Spring and other similar events raise important questions about the organization of the fear system in humans, ones which are amenable to study.
One question relates to the time course of the de-activation. Judging by events in the Arab Spring, it can be very long lasting indeed; indeed it may even be permanent. This may yet turn out to be an interesting example of brain-plasticity.
A second question relates to the trigger. There seems little reason to doubt that an emotional trigger was just that, a trigger coming on top of much discontent. This in turn raises the question whether there is a threshold for triggering an amygdalar de-activation, and what the neural mechanisms for maintaining it may be. It also raises the question of how the system returns to its de-fault value, assuming that the change in the brain is not permanent. Finally, it raises the question of how the two routes to the amygdala – the direct one and the cortical one – regulate one another.
A third question is about the potentiating effect on this de-activation caused by group action. There seems little doubt that the involvement of many in the uprising had a facilitating effect but how this works no one knows.
As I said above, the system regulating fear is not confined to the amygdala and an enquiry into the neural mechanisms involved in the loss of fear would no doubt have to extend much beyond it. But the amygdala is a good place to begin.
The Arab Spring is an example of other events where loss of fear is a powerful engine for initiating change. Hence the lessons that it provides for experimentation and the results of such experimentation will have important, general, implications.
The first is that this is a social network media revolution, through channels such as Facebook and Twitter. I find it hard to credit this oft-repeated belief. Both Tunisia (where the revolt started) and Egypt are very poor countries, 40% of the population in the latter living below the beltline of $2 per day. I do not suppose that, apart from a tiny percentage, many have the means to acquire computers and mobile ‘phones. Indeed it is hard to believe that Mohammad Bouazizi, the young man who tragically and in despair over his poverty immolated himself, had the means to have access to social media networks at all. Television, commonly available in cafés for communal watching, is likely to have played a more significant role. This is not to say that Twitter and Facebook did not facilitate communication. Of course they did, just as (in a much slower world) the horse facilitated communication during the French Revolution.
The second – loss of fear - is of greater significance and of much neurobiological interest. We have been repeatedly told that the masses who are revolting, commonly against much better equipped security forces, have lost their fear. Fear is associated with certain physiological activity, and especially prominent among brain structures contributing to such activity is a complex nucleus called the amygdala, buried within the temporal lobes of the brain, and consisting of many subdivisions. I would not wish to imply that the amygdala alone is responsible for so complex a state, for the amygdala is connected to many other brain structures which, collectively, are responsible for generating and maintaining the state of fear, as a defensive mechanism to protect the individual. Whatever the role of the different brain structures, the central role played by the amygdala was shown many years ago when scientists described how damage to it results in a loss of fear by animals and humans.
The amygdala has extensive connections within the brain. It is believed that there are two routes to the amygdala, an “immediate” one from the sense organs, which by-passes the cerebral cortex, and a more “leisurely” one that relays signals through the cerebral cortex. The amygdala is also connected to centres, such as those of the sympathetic nervous system, which regulate activity to mobilize the individual for appropriate reaction in response to fearful events or stimuli.
I suppose that the default state is activity – whatever its exact nature - within this system, activity within the amygdala that is relayed to other centres with which it is connected. I also assume, perhaps somewhat simplistically, that de-activation of this system is what leads to the condition that we describe as “loss of fear”.
This raises the interesting question of what triggers the de-activation and what dictates how long-lasting the de-activation and therefore the change from the default state is.
It is obvious that a long set of grievances reaches a point where individuals defy willfully the physiological state and care no longer about the consequences of their action. In the case of the Arab Spring, there was also an emotional trigger – the self immolation by Mohammad Bouazizi. The amygdala is part of the brain’s emotional and social system and the relative influence of the emotional component compared to the more cognitive component in regulating its activity is interesting to determine.
I am of the view that, in Egypt, a trigger, which was far more significant than Twitter and Facebook messages, was the emotional breakdown of Wael Ghoneim on Egyptian TV, an episode that was played and replayed, and presumably seen by masses in the many cafés in Egyptian cities. The episode was actually accompanied by music, hence fortifying the emotional message.
The fear system has been studied largely in animals, but the events of the Arab Spring and other similar events raise important questions about the organization of the fear system in humans, ones which are amenable to study.
One question relates to the time course of the de-activation. Judging by events in the Arab Spring, it can be very long lasting indeed; indeed it may even be permanent. This may yet turn out to be an interesting example of brain-plasticity.
A second question relates to the trigger. There seems little reason to doubt that an emotional trigger was just that, a trigger coming on top of much discontent. This in turn raises the question whether there is a threshold for triggering an amygdalar de-activation, and what the neural mechanisms for maintaining it may be. It also raises the question of how the system returns to its de-fault value, assuming that the change in the brain is not permanent. Finally, it raises the question of how the two routes to the amygdala – the direct one and the cortical one – regulate one another.
A third question is about the potentiating effect on this de-activation caused by group action. There seems little doubt that the involvement of many in the uprising had a facilitating effect but how this works no one knows.
As I said above, the system regulating fear is not confined to the amygdala and an enquiry into the neural mechanisms involved in the loss of fear would no doubt have to extend much beyond it. But the amygdala is a good place to begin.
The Arab Spring is an example of other events where loss of fear is a powerful engine for initiating change. Hence the lessons that it provides for experimentation and the results of such experimentation will have important, general, implications.
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